Quinn Yang

Expertise
Biotechnologies, Biomedical, Cellular Biology, Pathophysiology, Pharmacology, and Medicine.

Membership of Academic Societies
AAAS Member.
American Heart Association Basic Science Council Member.
American Physiology Society Member.

Education
Washington State University, Ph.D. Cardiovascular Physiology.
University of Montana, M.S. Pharmaceutical Sciences.
Guang Zhou University of TCM, Master of Medicine, Pediatrics.
Guang Zhou University of TCM, Bachelor of Medicine, Medicine.

Publications
Q. L. Yang: Study on treating allergic asthma in children with basic asthma formulas. In: Selected works of post-graduate thesis (1989), Guang Zhou College of TCM Press, Guang Zhou, 53-76.

Q. L. Yang, Y. C. Wu and X. Y. Zheng: Immunological and pharmacological studies on traditional herbs used to treat allergic asthma (Mini-review) J. of Guang Zhou College of TCM 7, 19-22. 1988.

Y. C. Wu, N. B. Li, W. M. Xu and Q. Yang: Influence of oxymatrine on releasing of allergic media in active immunized Guinea Pigs. Res. Ref. of Med. 12 (334), 54-62. 1990.

B. K. Slinker, R. L. Stephens, S. A. Fisher and Q. Yang. Immediate-early gene responses to different cardiac loads in the ejecting rabbit left ventricle. J. Mol. Cell. Cardiol. 28, 1565-1574. 1996.

Q.Yang, R. L. Stephens and B. K. Slinker: Mechanically-induced immediate-early gene expression in the isolated rabbit heart is partially mediated by angiotensin-II. J. Mol. Cell Cardiol. 28, A167. 1996.

Q. Yang, R. L. Stephens, J. W. Harding and B. K. Slinker: Angiotensin IV involvement in the mechanically induced Immediate-early gene expression in the isolated rabbit heart. Regulatory Peptides 71, 175-183,1997.

Q. Yang, A. Sanbe, H. E. Osinska, R. Klevisky, T. E. Hewett, and J. Robbins: A mouse model of myosin binding protein C human familial hypertrophic cardiomyopathy. J. of Clinical Investigation 102, 1292-1300, 1998. (Rapid publication).

Q. Yang, A. Sanbe, H. E. Osinska, R. Klevisky, T. E. Hewett and J. Robbins: In vivo modeling of myosin binding protein C familial hypertrophic cardiomyopathy. Circulation Research 85 (9): 841-847, 1999. (On the cover).

Q. Yang, T. E. Hewett, R. Klevisky, A. Sanbe, Xuejun Wang and J. Robbins: PKA dependent phosphorylation of Cardiac Myosin binding Protein C in transgenic mice. Cardiovasc Research 51(1):80-8, 2001.

Q. Yang, R. Klevisky and J. Robbins: Phenotypic deficits in mice expressing a Myosin Binding Protein C lacking the Titin and Myosin binding domains. Journal of Molecular and Cellular Cardiology. 2001 Sep;33(9):1649-58.

Lihong Cheng, Xiaowei Liu, Qianhong Qin, Xiao Yan, Patrick Smith, Jack Yu, Q. Yang: Hypertrophic stimuli induce cytosolic and nuclear accumulation of Arc in cardiomyocyte in vivo and in vitro (In preparation).

Lihong Cheng, Qianhong Qin, Guoliang Ding, Donna Floyd, Patrick Smith and Q. Yang: PKA phosphorylation at Serine284 of Cardiac Myosin Binding Protein C regulates cardiac diastolic function (In preparation).

Presentations
Oxytocin-induced stimulation of LH release: dependence on gonadal steroids. 1992, 52nd annual meeting of the Montana Academy of Sciences, Montana State University, Bozeman, MT, MBIN Section, talk #12 pg 17.

A mouse model of Familial Hypertrophic Cardiomyopathy caused by Myosin Binding protein-C mutation. The 71st Scientific session of American Heart Association annual meeting, November 11, 1998, Dallas Texas.

Structure-function studies of truncated cardiac myosin binding protein C in transgenic mice. Experimental Biology 99’, April 17-21, 1999, Washington DC.

Structure/function studies of hypertrophic cardiomyopathic mutations in transgenic mice. Molecular Biology of the Cardiovascular System, keystone Symposia, January 12-17, 2000. Snowbird, UT.

Invited Seminars
Cardiac Angiotensin System autoregulation in the heart. TCHRF, Children’s Hospital Medical Center, Cincinnati, OH. March 29, 1996.

Mouse Models of Human Familial Hypertrophic Cardiomyopathy with Mutations in Cardiac Myosin Binding Protein C. Division of Cardiology, Department of Pediatrics, Children’s Hospital Medical Center, Cincinnati, OH. April 6, 1998.

Structure-function studies of truncated cardiac myosin binding protein C in transgenic mice. Division of Cardiology, Department of Pediatrics, Children’s Hospital Medical Center, Cincinnati, OH. June 7, 1999 .

In vivo Modeling of Familial Hypertrophic Cardiomyopathy with mutations in cardiac myosin binding protein C. Cardiovascular Research Institute, Department of Surgery, University of Louisville School of Medicine, Louisville, KY. September 24, 1999 .

Transgenic mouse models of FHC linked to truncated forms of cardiac MyBP- C. Department of Physiology and Pharmacology, University of Medicine and Dentistry of New Jersey, Newark, NJ. October 29, 1999.

Structure/function studies of cMyBP-C and FHC. Laboratory of Physical Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, Methesda, MD. December 10, 1999.

Structure/function studies of cMyBP-C with a transgenic approach. Department of Veterinary and Comparative Anatomy, Pharmacology, and Physiology, Washington State University, Pullman, WA. January 27, 2000.

Structure/function studies of cMyBP-C in transgenic mice. Myogen Inc. Westminster, CO. April 21, 2000.

Structure/function studies of cardiac MyBP-C in transgenic mice. Cardiovascular Research Center, School of Medicine, University of Wisconsin at Madison, Madison , WI. May 31, 2000.

Myosin Binding Protein C in the Heart: from mutations to structural and functional defects, Department of Pathology, Emory University, Atlanta, Georgia, September 24, 2002.